SRA 2019 Postseason Power Rankings (in Three Words)
1. Cisek/Showalter - Tyler Cisek & Peter Jon Showatler (Amityville, NY & Columbus, OH)
Dominant, clutch, three-peat
2. Boysterous - Preston Bies & Caleb Heck (Santa Barbara, CA & Lancaster, PA)
Ambidextrous, doorstep, promising
3. Flexual Healing - Jarratt Rouse & Travis Core (Santa Barbara, CA & Cumming, GA)
Athletic, balanced, consistent
4. Mauktega - Kenny Ortega & Troy Mauk (San Diego, CA)
Scrappy, flashy, twins?
5. Trippy Lizard - Andrew Card & Clark Marshall (Chattanooga, TN)
Butter, tweeners, Fam
6. Spikers Synonymous - Tyler Stokes & Taylor Church (Provo, UT)
Spikeball Elite vindication
7. Anchored LI - Ryan Fitzgerald & Anthony Alvino (Long Island, NY)
Serveless, defenders, highlights
8. Graham/Throw - Joel Graham & Jesse Throw (Harrisonburg, VA & Chicago, IL)
Highs and lows
9. Chornaboles - Chris Hornacek & Skyler Boles (Chicago, IL & Fort Collins, CO)
Validated at nationals
10. Shake n Bake - David Gonzales & Matt Bohnen (Chattanooga, TN & Chicago, IL)
One hit wonder?
11. Club Spike Worldwide - Patrick Drucker & Chris Wilkins (Chicago, IL)
Definitely not Origin
12. Kahooters - Jacob Payer & Ryan Baillargeon (San Luis Obispo, CA & Los Angeles, CA)
Feet, serves, sand
13. Schweppes - Micah Zimmerman & Jesse Showalter (Lancaster, PA & Columbus, OH)
Experience, endurance, dives
14. Global Beans!!! - Coleman Epperson & Zac Vance (Chattanooga, TN & Newark, DE)
Yelling and screaming
15. KAPLOW!! - Gus Gonzales & Patric Chu (Athens, GA & Charleston, SC)
Lucky, then unlucky
16. Power Sweep - Addison Powers & Micah Corbett (Atlanta, GA)
Unlucky, then lucky
17. Elysium - Brian Childs & Logan Cornelius (Austin, TX & Dallas, TX)
True consolation champions
18. Team 7 - Thomas Hamilton & Spencer Weber (Livonia, MI)
Has some potential
19. Three Crowns - Sam Buckman & Warren Foote (San Luis Obispo, CA)
One hit wonder.
20. To Be Determined - Ravi Kandula & Emerson Dean (Providence, RI & Windsor, ON)
Alternate reality champs
21. Slice the Mango - Max Billinghurst & Dawson Morgan (Lansing, MI & Battle Creek, MI)
Finally did something
22. Strong Flush - Marc Etienne McLaughlin & Simon Brisebois (Montreal, QC)
Hurlant et crier
23. Sonic Boom - Ben Bunze & Connor Harte (Charlotte, NC & Chapel Hill, NC)
Made some flights
24. SubPar Team - Jacob Martinez & Josh Fragiacomo (Ventura, CA)
Should’ve trained more
25. ArcAngel - Fredric Hinkle & George Rizk (College Station, TX & Baltimore, MD)
Had some potential
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Scientists have found a new method of fighting antibiotic-resistant bacteria
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Stress often causes bacteria to form biofilms. Stress can manifest as a physical barrier, ultraviolet light, or a toxic substance such as antibiotics. The formation of these biofilms takes from several hours to days, and they can be of different shapes, sizes, colors, and textures depending on the type of bacteria. Being in a biofilm state protects them from harmful substances in the environment – biofilms have a unique outer wall with different physical and chemical properties than their individual cells. They can coordinate metabolism, slow down their growth, and even form an impenetrable barrier of wrinkles and creases. This is one of the ways they achieve high antibiotic resistance. Researchers from the United Kingdom recently studied the transition of the Hay Bacillus bacterium from a free-moving swarm to a biofilm as a defense mechanism and published what they did to combat its antibiotic-resistant properties in eLife.
To determine if their test strain was behaving like the others, they first ran stress tests on them. They tested the bacteria’s response to the physical barrier, ultraviolet light, and antibiotics. The addition of a physical barrier led to the transition of bacteria from one layer to a multilayer layer, followed by an increase in cell density and the formation of multilayer islands near the barrier. Later, wrinkles formed on the islands near the barrier in the place where they began to appear initially.
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When they applied ultraviolet light to the swarm, they again observed a drop in cell speed and an increase in density. And after the scientists added a large dose of the antibiotic kanamycin, the bacterial cells formed a biofilm. The researchers then developed a strategy to combat this bacterial biofilm. They added kanamycin to the environment of a new batch of swarming bacterial cells and watched as the biofilm began to form. They then re-injected the antibiotic at a much higher dose than the first, just before the biofilm formation was complete. As a result, the partially formed biofilm was destroyed and bacterial cells died. This shows that antibiotic-resistant bacteria lose their resistance to antibiotics when they undergo a phase transition, right before going into the biofilm, where they will become much more resistant. Thus, with the correct administration of antibiotics, the bacteria can be attacked in their most vulnerable state and destroyed. The researchers believe that similar transitions from swarm to biofilm occur in other bacterial species. Their research may pave the way for finding more effective ways to control clinically relevant bacteria. For example, Salmonella enterica, which spreads into the bloodstream and is transmitted through contaminated food. Or Pseudomonas aeruginosa with multiple drug resistance, which after surgery causes infections in the blood, lungs (pneumonia) and other parts of the body and spreads in hospitals.
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Scientists have found a new method of fighting antibiotic-resistant bacteria
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Stress often causes bacteria to form biofilms. Stress can manifest as a physical barrier, ultraviolet light, or a toxic substance such as antibiotics. The formation of these biofilms takes from several hours to days, and they can be of different shapes, sizes, colors, and textures depending on the type of bacteria. Being in a biofilm state protects them from harmful substances in the environment – biofilms have a unique outer wall with different physical and chemical properties than their individual cells. They can coordinate metabolism, slow down their growth, and even form an impenetrable barrier of wrinkles and creases. This is one of the ways they achieve high antibiotic resistance. Researchers from the United Kingdom recently studied the transition of the Hay Bacillus bacterium from a free-moving swarm to a biofilm as a defense mechanism and published what they did to combat its antibiotic-resistant properties in eLife.
To determine if their test strain was behaving like the others, they first ran stress tests on them. They tested the bacteria’s response to the physical barrier, ultraviolet light, and antibiotics. The addition of a physical barrier led to the transition of bacteria from one layer to a multilayer layer, followed by an increase in cell density and the formation of multilayer islands near the barrier. Later, wrinkles formed on the islands near the barrier in the place where they began to appear initially.
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When they applied ultraviolet light to the swarm, they again observed a drop in cell speed and an increase in density. And after the scientists added a large dose of the antibiotic kanamycin, the bacterial cells formed a biofilm. The researchers then developed a strategy to combat this bacterial biofilm. They added kanamycin to the environment of a new batch of swarming bacterial cells and watched as the biofilm began to form. They then re-injected the antibiotic at a much higher dose than the first, just before the biofilm formation was complete. As a result, the partially formed biofilm was destroyed and bacterial cells died. This shows that antibiotic-resistant bacteria lose their resistance to antibiotics when they undergo a phase transition, right before going into the biofilm, where they will become much more resistant. Thus, with the correct administration of antibiotics, the bacteria can be attacked in their most vulnerable state and destroyed. The researchers believe that similar transitions from swarm to biofilm occur in other bacterial species. Their research may pave the way for finding more effective ways to control clinically relevant bacteria. For example, Salmonella enterica, which spreads into the bloodstream and is transmitted through contaminated food. Or Pseudomonas aeruginosa with multiple drug resistance, which after surgery causes infections in the blood, lungs (pneumonia) and other parts of the body and spreads in hospitals.
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Scientists have found a new method of fighting antibiotic-resistant bacteria
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Stress often causes bacteria to form biofilms. Stress can manifest as a physical barrier, ultraviolet light, or a toxic substance such as antibiotics. The formation of these biofilms takes from several hours to days, and they can be of different shapes, sizes, colors, and textures depending on the type of bacteria. Being in a biofilm state protects them from harmful substances in the environment – biofilms have a unique outer wall with different physical and chemical properties than their individual cells. They can coordinate metabolism, slow down their growth, and even form an impenetrable barrier of wrinkles and creases. This is one of the ways they achieve high antibiotic resistance. Researchers from the United Kingdom recently studied the transition of the Hay Bacillus bacterium from a free-moving swarm to a biofilm as a defense mechanism and published what they did to combat its antibiotic-resistant properties in eLife.
To determine if their test strain was behaving like the others, they first ran stress tests on them. They tested the bacteria’s response to the physical barrier, ultraviolet light, and antibiotics. The addition of a physical barrier led to the transition of bacteria from one layer to a multilayer layer, followed by an increase in cell density and the formation of multilayer islands near the barrier. Later, wrinkles formed on the islands near the barrier in the place where they began to appear initially.
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When they applied ultraviolet light to the swarm, they again observed a drop in cell speed and an increase in density. And after the scientists added a large dose of the antibiotic kanamycin, the bacterial cells formed a biofilm. The researchers then developed a strategy to combat this bacterial biofilm. They added kanamycin to the environment of a new batch of swarming bacterial cells and watched as the biofilm began to form. They then re-injected the antibiotic at a much higher dose than the first, just before the biofilm formation was complete. As a result, the partially formed biofilm was destroyed and bacterial cells died. This shows that antibiotic-resistant bacteria lose their resistance to antibiotics when they undergo a phase transition, right before going into the biofilm, where they will become much more resistant. Thus, with the correct administration of antibiotics, the bacteria can be attacked in their most vulnerable state and destroyed. The researchers believe that similar transitions from swarm to biofilm occur in other bacterial species. Their research may pave the way for finding more effective ways to control clinically relevant bacteria. For example, Salmonella enterica, which spreads into the bloodstream and is transmitted through contaminated food. Or Pseudomonas aeruginosa with multiple drug resistance, which after surgery causes infections in the blood, lungs (pneumonia) and other parts of the body and spreads in hospitals.
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